Scientists Can Not Determine The Cause Of Autism

Scientists Can Not Determine The Cause Of Autism.
Some children who are diagnosed with autism at an untimely maturity will ultimately shed all signs and symptoms of the untidiness as they enter adolescence or young adulthood, a new analysis contends. Whether that happens because of aggressive interventions or whether it boils down to biology and genetics is still unclear, the researchers noted, although experts doubt it is most likely a set of the two. The finding stems from a methodical analysis of 34 children who were deemed "normal" at the study's start, ignoring having been diagnosed with autism before the age of 5.

So "Generally, autism is looked at as a lifelong disorder," said mug up author Deborah Fein, a professor in the departments of behaviour and pediatrics at the University of Connecticut. "The point of this work was really to demonstrate and chronicle this phenomenon, in which some children can move off the autism spectrum and really go on to function like normal adolescents in all areas, and end up mainstreamed in automatic classrooms with no one-on-one support.

And "Although we don't know unerringly what percent of these kids are capable of this kind of amazing outcome, we do know it's a minority. We're certainly talking about less than 25 percent of those diagnosed with autism at an near the start age. "Certainly all autistic children can get better and blossom with good therapy. But this is not just about good therapy. I've seen thousands of kids who have great group therapy but don't reach this result. It's very, very important that parents who don't keep company with this outcome not feel as if they did something wrong".

Fein and her colleagues reported the findings of their study, which was supported by the US National Institutes of Health, in the Jan. 15 young of the Journal of Child Psychology and Psychiatry. The 34 individuals at one time diagnosed with autism (most between the ages of 2 and 4) were inefficiently between the ages of 8 and 21 during the study. They were compared to a group of 44 individuals with high-functioning autism and a device group of 34 "normal" peers.

In-depth blind analysis of each child's underived diagnostic report revealed that the now-"optimal outcome" group had, as young children, shown signs of group impairment that was milder than the 44 children who had "high-functioning" autism. As litter children, the now-optimal group had suffered from equally severe communication impairment and repetitive behaviors as those in the high-functioning group.