Dangerous Bacteria Live On Chicken Breasts.
Potentially poisonous bacteria was found on 97 percent of chicken breasts bought at stores across the United States and tested, according to a reborn work in Dec 2013. And about half of the chicken samples had at least one category of bacteria that was resistant to three or more classes of antibiotics, the investigators found. The tests on the 316 untrained chicken breasts also found that most had bacteria - such as enterococcus and E coli - linked to fecal contamination.
About 17 percent of the E coli were a breed that can cause urinary tract infections, according to the study, published online and in the February 2014 question of Consumer Reports. In addition, slight more than 11 percent had two or more types of multidrug-resistant bacteria. Bacteria on the chicken were more averse to antibiotics used to promote chicken growth and to prevent poultry diseases than to other types of antibiotics, the on found.
These findings show that "consumers who buy chicken breast at their local grocery stores are very plausible to get a sample that is contaminated and likely to get a bug that is multi-drug resistant. When people get psychoneurotic from resistant bacteria, treatment may be getting harder to find," said Dr Urvashi Rangan, a toxicologist and administration director of the Food Safety and Sustainability Center at Consumer Reports. The publication has been testing US chicken since 1998, and rates of contamination with salmonella have not changed much during that time, ranging from 11 percent to 16 percent of samples.
Showing posts with label bacteria. Show all posts
Showing posts with label bacteria. Show all posts
Thursday 2 May 2019
Tuesday 25 September 2018
People Carries A Few Hundred Types Of Bacteria
People Carries A Few Hundred Types Of Bacteria.
If you were to change-over from vegetarianism to meat-eating, or vice-versa, chances are the configuration of your gut bacteria would also undergo a big change, a restored study suggests. The research, published Dec 11, 2013 in the record Nature, showed that the number and kinds of bacteria - and even the way the bacteria behaved - changed within a lifetime of switching from a normal diet to eating either animal- or plant-based foods exclusively. "Not only were there changes in the over-abundance of different bacteria, but there were changes in the kinds of genes that they were expressing and their activity," said lessons author Lawrence David, an assistant professor at the Institute for Genome Sciences and Policy at Duke University.
Trillions of bacteria unexploded in each person's gut. They're thought to play a job in digestion, immunity and possibly even body weight. The study suggests that this bacterial community and its genes - called the microbiome - are extraordinarily compliant and capable of responding swiftly to whatever is coming its way. "The basic microbiome is potentially quite sensitive to what we eat. And it is delicate on time scales shorter than had previously been thought, however, that it's hard to pick on out exactly what that might mean for human health.
Another expert agreed. "It's nice to have some solid substantiation now that these types of significant changes in diet can impact the gut microflora in a significant way," said Jeffrey Cirillo, a professor of microbial and molecular pathogenesis at the Texas Aandamp;M Health Science Center College of Medicine in Bryan, Texas. "That's very cordial to see, and it's very rapid. It's surprising how expeditious the changes can occur".
If you were to change-over from vegetarianism to meat-eating, or vice-versa, chances are the configuration of your gut bacteria would also undergo a big change, a restored study suggests. The research, published Dec 11, 2013 in the record Nature, showed that the number and kinds of bacteria - and even the way the bacteria behaved - changed within a lifetime of switching from a normal diet to eating either animal- or plant-based foods exclusively. "Not only were there changes in the over-abundance of different bacteria, but there were changes in the kinds of genes that they were expressing and their activity," said lessons author Lawrence David, an assistant professor at the Institute for Genome Sciences and Policy at Duke University.
Trillions of bacteria unexploded in each person's gut. They're thought to play a job in digestion, immunity and possibly even body weight. The study suggests that this bacterial community and its genes - called the microbiome - are extraordinarily compliant and capable of responding swiftly to whatever is coming its way. "The basic microbiome is potentially quite sensitive to what we eat. And it is delicate on time scales shorter than had previously been thought, however, that it's hard to pick on out exactly what that might mean for human health.
Another expert agreed. "It's nice to have some solid substantiation now that these types of significant changes in diet can impact the gut microflora in a significant way," said Jeffrey Cirillo, a professor of microbial and molecular pathogenesis at the Texas Aandamp;M Health Science Center College of Medicine in Bryan, Texas. "That's very cordial to see, and it's very rapid. It's surprising how expeditious the changes can occur".
Monday 16 July 2018
Awareness Against The Global Problem Of Antibiotic Resistance
Awareness Against The Global Problem Of Antibiotic Resistance.
Knowing when to tolerate antibiotics - and when not to - can servant fight the rise of deadly "superbugs," impart experts at the US Centers for Disease Control and Prevention. About half of antibiotics prescribed are non-essential or inappropriate, the agency says, and overuse has helped create bacteria that don't respond, or rejoin less effectively, to the drugs used to fight them. "Antibiotics are a shared resource that has become a rare resource," said Dr Lauri Hicks, a medical epidemiologist at the CDC.
She's also medical top banana a of new program, Get Smart: Know When Antibiotics Work, that had its launch this week. "Everyone has a lines to play in preventing the spread of antibiotic resistance". The stakes are high, said Dr Arjun Srinivasan, CDC's mate director for health care-associated infection restraining programs. Almost every type of bacteria has become stronger and less responsive to antibiotic treatment.
The CDC is urging Americans to use the drugs nicely to help prevent the global problem of antibiotic resistance. To that end, the US Food and Drug Administration (FDA), numerous nationalistic medical and methodical associations, as well as state and local health departments have collaborated on the CDC's Get Smart initiative.
Most strains of antibiotic-resistant bacteria are still found in condition care settings, such as hospitals and nursing homes. Yet superbugs, including MRSA (methicillin-resistant staphylococcus aureus) - which kills about 19000 Americans a year - are increasingly found in community settings, such as vigour clubs, schools, and workplaces, said Hicks.
Community-associated MRSA (CA-MRSA), a impair that affects fine fettle people outside of hospitals, made headlines in 2008, when it killed a Florida turned on school football player. Referring to late reports of sinusitis caused by MRSA, Hicks said that "people who would normally be treated with an spoken antibiotic are requiring more toxic medications or, in some instances, admission to a hospital. We've seen this with pneumonia, too, and I nails we'll start to see it with other types of infections as well".
Knowing when to tolerate antibiotics - and when not to - can servant fight the rise of deadly "superbugs," impart experts at the US Centers for Disease Control and Prevention. About half of antibiotics prescribed are non-essential or inappropriate, the agency says, and overuse has helped create bacteria that don't respond, or rejoin less effectively, to the drugs used to fight them. "Antibiotics are a shared resource that has become a rare resource," said Dr Lauri Hicks, a medical epidemiologist at the CDC.
She's also medical top banana a of new program, Get Smart: Know When Antibiotics Work, that had its launch this week. "Everyone has a lines to play in preventing the spread of antibiotic resistance". The stakes are high, said Dr Arjun Srinivasan, CDC's mate director for health care-associated infection restraining programs. Almost every type of bacteria has become stronger and less responsive to antibiotic treatment.
The CDC is urging Americans to use the drugs nicely to help prevent the global problem of antibiotic resistance. To that end, the US Food and Drug Administration (FDA), numerous nationalistic medical and methodical associations, as well as state and local health departments have collaborated on the CDC's Get Smart initiative.
Most strains of antibiotic-resistant bacteria are still found in condition care settings, such as hospitals and nursing homes. Yet superbugs, including MRSA (methicillin-resistant staphylococcus aureus) - which kills about 19000 Americans a year - are increasingly found in community settings, such as vigour clubs, schools, and workplaces, said Hicks.
Community-associated MRSA (CA-MRSA), a impair that affects fine fettle people outside of hospitals, made headlines in 2008, when it killed a Florida turned on school football player. Referring to late reports of sinusitis caused by MRSA, Hicks said that "people who would normally be treated with an spoken antibiotic are requiring more toxic medications or, in some instances, admission to a hospital. We've seen this with pneumonia, too, and I nails we'll start to see it with other types of infections as well".
Saturday 1 July 2017
Some Bacteria Inhibit Cancer Progression
Some Bacteria Inhibit Cancer Progression.
Having a discount variety of bacteria in the despoil is associated with colorectal cancer, according to a new study. Researchers analyzed DNA in fecal samples unruffled from 47 colorectal cancer patients and 94 people without the disease to decide the level of diversity of their gut bacteria. Study authors led by Jiyoung Ahn, at the New York University School of Medicine, concluded that decreased bacterial variation in the gut was associated with colorectal cancer.
The sanctum was published in the Dec 6, 2013 issue of the Journal of the National Cancer Institute. Colorectal cancer patients had put down levels of bacteria that ferment dietary fiber into butyrate. This fatty acid may repress inflammation and the start of cancer in the colon, researchers found.
Having a discount variety of bacteria in the despoil is associated with colorectal cancer, according to a new study. Researchers analyzed DNA in fecal samples unruffled from 47 colorectal cancer patients and 94 people without the disease to decide the level of diversity of their gut bacteria. Study authors led by Jiyoung Ahn, at the New York University School of Medicine, concluded that decreased bacterial variation in the gut was associated with colorectal cancer.
The sanctum was published in the Dec 6, 2013 issue of the Journal of the National Cancer Institute. Colorectal cancer patients had put down levels of bacteria that ferment dietary fiber into butyrate. This fatty acid may repress inflammation and the start of cancer in the colon, researchers found.
Friday 30 June 2017
Excessive Use Of Antibiotics In Animal Husbandry Creates A Deadly Intestinal Bacteria
Excessive Use Of Antibiotics In Animal Husbandry Creates A Deadly Intestinal Bacteria.
The theme of E coli bacteria that this month killed dozens of rank and file in Europe and sickened thousands more may be more harmful because of the way it has evolved, a new review suggests. Scientists say this strain of E coli produces a particularly noxious toxin and also has a good ability to hold on to cells within the intestine. This, alongside the fact that it is also resistant to many antibiotics, has made the soi-disant O104:H4 strain both deadlier and easier to transmit, German researchers report.
And "This exertion of E coli is much nastier than its more common cousin E coli O157, which is unclean enough - about three times more virulent," said Hugh Pennington, emeritus professor of bacteriology at the University of Aberdeen in Scotland and writer of an accompanying editorial published online June 23, 2011 in The Lancet Infectious Diseases. Another study, published the same date in the New England Journal of Medicine, concludes that, as of June 18, 2011, more than 3200 clan have fallen hostile in Germany due to the outbreak, including 39 deaths.
In fact, the German extraction - traced to sprouts raised at a German organic farm - "was reliable for the deadliest E coli outbreak in history. It may well be so nasty because it combines the virulence factors of shiga toxin, produced by E coli O157, and the structure for sticking to intestinal cells cast-off by another strain of E coli, enteroaggregative E coli, which is known to be an important cause of diarrhea in poorer countries".
Shiga toxin can also daily spur what doctors call "hemolytic uremic syndrome," a potentially devastating form of kidney failure. In the New England Journal of Medicine study, German researchers estimate that 25 percent of outbreak cases involved this complication. The bottom line, according to Pennington: "E coli hasn't gone away. It still springs surprises".
To distinguish out how this thread of the intestinal bug proved so lethal, researchers led by Dr Helge Karch from the University of Munster wilful 80 samples of the bacteria from affected patients. They tested the samples for shiga toxin-producing E coli and also for acerbity genes of other types of E coli.
The theme of E coli bacteria that this month killed dozens of rank and file in Europe and sickened thousands more may be more harmful because of the way it has evolved, a new review suggests. Scientists say this strain of E coli produces a particularly noxious toxin and also has a good ability to hold on to cells within the intestine. This, alongside the fact that it is also resistant to many antibiotics, has made the soi-disant O104:H4 strain both deadlier and easier to transmit, German researchers report.
And "This exertion of E coli is much nastier than its more common cousin E coli O157, which is unclean enough - about three times more virulent," said Hugh Pennington, emeritus professor of bacteriology at the University of Aberdeen in Scotland and writer of an accompanying editorial published online June 23, 2011 in The Lancet Infectious Diseases. Another study, published the same date in the New England Journal of Medicine, concludes that, as of June 18, 2011, more than 3200 clan have fallen hostile in Germany due to the outbreak, including 39 deaths.
In fact, the German extraction - traced to sprouts raised at a German organic farm - "was reliable for the deadliest E coli outbreak in history. It may well be so nasty because it combines the virulence factors of shiga toxin, produced by E coli O157, and the structure for sticking to intestinal cells cast-off by another strain of E coli, enteroaggregative E coli, which is known to be an important cause of diarrhea in poorer countries".
Shiga toxin can also daily spur what doctors call "hemolytic uremic syndrome," a potentially devastating form of kidney failure. In the New England Journal of Medicine study, German researchers estimate that 25 percent of outbreak cases involved this complication. The bottom line, according to Pennington: "E coli hasn't gone away. It still springs surprises".
To distinguish out how this thread of the intestinal bug proved so lethal, researchers led by Dr Helge Karch from the University of Munster wilful 80 samples of the bacteria from affected patients. They tested the samples for shiga toxin-producing E coli and also for acerbity genes of other types of E coli.
Wednesday 25 January 2017
New Treatments For Asthma
New Treatments For Asthma.
Researchers reply they've discovered why infants who tangible in homes with a dog are less likely to develop asthma and allergies later in childhood. The span conducted experiments with mice and found that exposing them to dust from homes where dogs live triggered changes in the community of microbes that material in the infant's gut and reduced immune system answer to common allergens. The scientists also identified a specific species of gut bacteria that's important in protecting the airways against allergens and viruses that cause respiratory infections, according to the study published online Dec 16, 2013 in the Proceedings of the National Academy of Sciences.
While these findings were made in mice, they're also odds-on to interpret why children who are exposed to dogs from the time they're born are less seemly to have allergies and asthma, the University of California, San Francisco (UCSF) and University of Michigan researchers said. These results also suggest that changes in the sack bacteria community (gut microbiome) can impress immune function elsewhere in the body, said study co-leader Susan Lynch, an accessory professor in the gastroenterology division at UCSF.
Researchers reply they've discovered why infants who tangible in homes with a dog are less likely to develop asthma and allergies later in childhood. The span conducted experiments with mice and found that exposing them to dust from homes where dogs live triggered changes in the community of microbes that material in the infant's gut and reduced immune system answer to common allergens. The scientists also identified a specific species of gut bacteria that's important in protecting the airways against allergens and viruses that cause respiratory infections, according to the study published online Dec 16, 2013 in the Proceedings of the National Academy of Sciences.
While these findings were made in mice, they're also odds-on to interpret why children who are exposed to dogs from the time they're born are less seemly to have allergies and asthma, the University of California, San Francisco (UCSF) and University of Michigan researchers said. These results also suggest that changes in the sack bacteria community (gut microbiome) can impress immune function elsewhere in the body, said study co-leader Susan Lynch, an accessory professor in the gastroenterology division at UCSF.
Wednesday 6 April 2016
Scientists Have Discovered A New Kind Of Staphylococcus
Scientists Have Discovered A New Kind Of Staphylococcus.
Potentially destructive staph bacteria can wait deep inside the nose, a small new ponder finds. Researchers tested 12 healthy people and found that formerly overlooked sites perspicacious within the nose may be reservoirs for Staphylococcus aureus, which is a major cause of disease. Nearly half of S aureus strains are antibiotic-resistant. It's been known that S aureus can reside on the overlay and at sites discount down in the nose.
Although there are ways to eliminate the bacteria, it typically returns in weeks or months. This uncharted finding that the bacteria can be present further inside the nose may explain why this happens, the Stanford University School of Medicine researchers said. "About one-third of all consumers are persistent S aureus carriers, another third are accessory carriers and a remaining third don't seem to carry S aureus at all," swat senior author Dr David Relman, a professor of medicine and microbiology and immunology, said in a university dirt release.
Potentially destructive staph bacteria can wait deep inside the nose, a small new ponder finds. Researchers tested 12 healthy people and found that formerly overlooked sites perspicacious within the nose may be reservoirs for Staphylococcus aureus, which is a major cause of disease. Nearly half of S aureus strains are antibiotic-resistant. It's been known that S aureus can reside on the overlay and at sites discount down in the nose.
Although there are ways to eliminate the bacteria, it typically returns in weeks or months. This uncharted finding that the bacteria can be present further inside the nose may explain why this happens, the Stanford University School of Medicine researchers said. "About one-third of all consumers are persistent S aureus carriers, another third are accessory carriers and a remaining third don't seem to carry S aureus at all," swat senior author Dr David Relman, a professor of medicine and microbiology and immunology, said in a university dirt release.
Wednesday 8 April 2015
The Multiple Sclerosis Risk Factors
The Multiple Sclerosis Risk Factors.
Women who harbor the longing bacteria Helicobacter pylori (or H pylori) may be less plausible to develop multiple sclerosis (MS), a redone study suggests. In the study, researchers found that among women with MS - an often disabling infirmity of the central nervous system - 14 percent had evidence of dead infection with H pylori. But 22 percent of healthy women in the study had denote of a previous H pylori infection. H pylori bacteria settle in the gut, and while the craze usually causes no problems, it can eventually lead to ulcers or even stomach cancer. It's estimated that half of the world's natives carries H pylori, but the prevalence is much lower in wealthier countries than developing ones, according to CV information in the study.
And "Helicobacter is typically acquired in childhood and correlates in a with hygiene," explained Dr Allan Kermode, the senior researcher on the new review and a professor of neurology at the University of Western Australia in Perth. The reason for the connection between H pylori and MS isn't clear, and researchers only found an association, not a cause-and-effect link. But Kermode said his scrutinize supports the theory that settled infections early in life might curb the gamble of MS later on - which means the increasingly hygienic surroundings in developed countries could have a downside.
So "It's plausible," agreed Bruce Bebo, administrator vice-president of research for the National Multiple Sclerosis Society in New York City. "The theory is, our trendy immune methodology may be more susceptible to developing autoimmune disease". Multiple sclerosis is thought to arise when the immune process mistakenly attacks the protective sheath around nerve fibers in the brain and spine, according to an editorial published with the survey on Jan 19, 2015 in the Journal of Neurology, Neurosurgery and Psychiatry.
No one knows what triggers that aberrant immune response. But according to the "hygiene hypothesis," Bebo explained, early obsession encounters with bacteria and other bugs may help steer the immune system into disease-fighting mode - and away from attacks on the body's tonic tissue. So, people who have not been exposed to common pathogens, counterpart H pylori, might be at increased risk of autoimmune diseases like MS.
Women who harbor the longing bacteria Helicobacter pylori (or H pylori) may be less plausible to develop multiple sclerosis (MS), a redone study suggests. In the study, researchers found that among women with MS - an often disabling infirmity of the central nervous system - 14 percent had evidence of dead infection with H pylori. But 22 percent of healthy women in the study had denote of a previous H pylori infection. H pylori bacteria settle in the gut, and while the craze usually causes no problems, it can eventually lead to ulcers or even stomach cancer. It's estimated that half of the world's natives carries H pylori, but the prevalence is much lower in wealthier countries than developing ones, according to CV information in the study.
And "Helicobacter is typically acquired in childhood and correlates in a with hygiene," explained Dr Allan Kermode, the senior researcher on the new review and a professor of neurology at the University of Western Australia in Perth. The reason for the connection between H pylori and MS isn't clear, and researchers only found an association, not a cause-and-effect link. But Kermode said his scrutinize supports the theory that settled infections early in life might curb the gamble of MS later on - which means the increasingly hygienic surroundings in developed countries could have a downside.
So "It's plausible," agreed Bruce Bebo, administrator vice-president of research for the National Multiple Sclerosis Society in New York City. "The theory is, our trendy immune methodology may be more susceptible to developing autoimmune disease". Multiple sclerosis is thought to arise when the immune process mistakenly attacks the protective sheath around nerve fibers in the brain and spine, according to an editorial published with the survey on Jan 19, 2015 in the Journal of Neurology, Neurosurgery and Psychiatry.
No one knows what triggers that aberrant immune response. But according to the "hygiene hypothesis," Bebo explained, early obsession encounters with bacteria and other bugs may help steer the immune system into disease-fighting mode - and away from attacks on the body's tonic tissue. So, people who have not been exposed to common pathogens, counterpart H pylori, might be at increased risk of autoimmune diseases like MS.
Sunday 1 March 2015
A New Antibiotic For Fighting Disease-Causing Bacteria
A New Antibiotic For Fighting Disease-Causing Bacteria.
Laboratory researchers roughly they've discovered a additional antibiotic that could prove valuable in fighting disease-causing bacteria that no longer answer to older, more frequently used drugs. The new antibiotic, teixobactin, has proven true against a number of bacterial infections that have developed resistance to existing antibiotic drugs, researchers write-up in Jan 7, 2015 in the journal Nature. Researchers have used teixobactin to prescription lab mice of MRSA (methicillin-resistant Staphylococcus aureus), a bacterial infection that sickens 80000 Americans and kills 11000 every year, according to the US Centers for Disease Control and Prevention (CDC).
The creative antibiotic also worked against the bacteria that causes pneumococcal pneumonia. Cell enlightenment tests also showed that the uncharted drug effectively killed off drug-resistant strains of tuberculosis, anthrax and Clostridium difficile, a bacteria that causes life-threatening diarrhea and is associated with 250000 infections and 14000 deaths in the United States each year, according to the CDC. "My appraise is that we will unquestionably be in clinical trials three years from now," said the study's elder author, Kim Lewis, director of the Antimicrobial Discovery Center at Northeastern University in Boston.
Lewis said researchers are working to focus the inexperienced antibiotic and make it more effective for use in humans. Dr Ambreen Khalil, an infectious disease connoisseur at Staten Island University Hospital in New York City, said teixobactin "has the covert of being a valuable addition to a limited number of antibiotic options that are currently available". In particular, its effectiveness against MRSA "may corroborate to be critically significant".
And its potent activity against C difficile also "makes it a propitious compound at this time". Most antibiotics are created from bacteria found in the soil, but only about 1 percent of these microorganisms will blossom in petri dishes in laboratories. Because of this, it's become increasingly fussy to find new antibiotics in nature. The 1960s heralded the end of the original era of antibiotic discovery, and synthetic antibiotics were unable to replace natural products, the authors said in distance notes.
Laboratory researchers roughly they've discovered a additional antibiotic that could prove valuable in fighting disease-causing bacteria that no longer answer to older, more frequently used drugs. The new antibiotic, teixobactin, has proven true against a number of bacterial infections that have developed resistance to existing antibiotic drugs, researchers write-up in Jan 7, 2015 in the journal Nature. Researchers have used teixobactin to prescription lab mice of MRSA (methicillin-resistant Staphylococcus aureus), a bacterial infection that sickens 80000 Americans and kills 11000 every year, according to the US Centers for Disease Control and Prevention (CDC).
The creative antibiotic also worked against the bacteria that causes pneumococcal pneumonia. Cell enlightenment tests also showed that the uncharted drug effectively killed off drug-resistant strains of tuberculosis, anthrax and Clostridium difficile, a bacteria that causes life-threatening diarrhea and is associated with 250000 infections and 14000 deaths in the United States each year, according to the CDC. "My appraise is that we will unquestionably be in clinical trials three years from now," said the study's elder author, Kim Lewis, director of the Antimicrobial Discovery Center at Northeastern University in Boston.
Lewis said researchers are working to focus the inexperienced antibiotic and make it more effective for use in humans. Dr Ambreen Khalil, an infectious disease connoisseur at Staten Island University Hospital in New York City, said teixobactin "has the covert of being a valuable addition to a limited number of antibiotic options that are currently available". In particular, its effectiveness against MRSA "may corroborate to be critically significant".
And its potent activity against C difficile also "makes it a propitious compound at this time". Most antibiotics are created from bacteria found in the soil, but only about 1 percent of these microorganisms will blossom in petri dishes in laboratories. Because of this, it's become increasingly fussy to find new antibiotics in nature. The 1960s heralded the end of the original era of antibiotic discovery, and synthetic antibiotics were unable to replace natural products, the authors said in distance notes.
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